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Neuroprotektive Wirkung von N6- Isopentanyl Adenosin

Neuroprotektive Wirkung von N6- Isopentanyl Adenosin

Anand VijayaKumar Parul Ramkrishnan / Kumari Sonal Gupta / Sangmeshwar Baburao Shinde

73,52 €
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Editorial:
KS OmniScriptum Publishing
Año de edición:
2026
Materia
Farmacología
ISBN:
9786630082180
73,52 €
IVA incluido
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Sobre IBD

Die Alzheimer-Krankheit (AK), die häufigste Form der Demenz, ist eine fortschreitende neurodegenerative Erkrankung, von der Millionen Menschen, insbesondere in der alternden Bevölkerung, betroffen sind. Gekennzeichnet durch senile Plaques, Amyloid-Beta (Aβ)-Ablagerungen, neurofibrilläre Tangles und selektiven neuronalen Verlust, führt die AK zu einem allmählichen Rückgang der kognitiven Fähigkeiten, des Gedächtnisses und der alltäglichen Funktionen. Während aktuelle Behandlungen in erster Linie auf eine symptomatische Linderung durch Modulation von Neurotransmitterspiegeln abzielen, tragen sie kaum dazu bei, das Fortschreiten der Krankheit aufzuhalten.N6-Isopentenyladenosin (iPA), ein natürlich vorkommendes Purinderivat, ist ein vielversprechender neuer therapeutischer Ansatz bei neurodegenerativen Erkrankungen wie Alzheimer. Mit seinen bemerkenswerten entzündungshemmenden, antioxidativen und antiapoptotischen Eigenschaften moduliert iPA nachweislich wichtige Signalwege, die das Überleben und die Plastizität von Nervenzellen fördern. Präklinische Studien haben sein Potenzial gezeigt, Entzündungen zu reduzieren, neuronale Schäden zu mildern und die kognitiven Funktionen in Tiermodellen zu verbessern, was neue Hoffnung für AK-Patienten bietet.

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